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Danilon (Norfloxacin)
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Norfloxacin

Danilon is an antibiotic in a group of drugs called fluoroquinolones (flor-o-KWIN-o-lones). Danilon fights bacteria in the body. Danilon is used to treat different bacterial infections of the prostate or urinary tract (bladder and kidneys). Danilon is also used to treat gonorrhea.

Other names for this medication:
Norfloxacin, Noroxin, Ambigram, Baccidal, Barazan, Bexinor, Chibroxin, Chibroxine, Danilon, Epinor, Firin, Flox, Floxatral, Grenis, Janacin, Lexinor, Loxone, Microxin, Nalion, Noflo, Nolicin, Noprose, Noracin, Norax, Norbactin, Norflohexal, Norilet, Normax, Noroxine, Norsol, Norzen, Nufloxib, Oranor, Renor, Respexil, Senro, Shinun, Trizolin, Uritracin, Uroxacin, Utinor, Zoroxin,

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Immunochemical crossreactivity between globulins from buckwheat and indigo seeds. Characterisation of chemical components for identifying historical Chinese textile dyes by ultra high performance liquid chromatography - photodiode array - electrospray ionisation mass spectrometer. We hypothesized that reconstruction of the central band of the IOLC in cadaver forearms using a Mini-TightRope suture-button construct would restore native forearm stability.

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High-mobility two-dimensional electron gas in SrGeO3- and BaSnO3-based perovskite oxide heterostructures: an ab initio study. Gadolinium (Gd)(III), which has seven unpaired electrons and a large magnetic moment, can dramatically influence the water proton relaxation and hence exhibits excellent MRI contrast. Minimally invasive gynaecological surgeries are performed for several malignant and nonmalignant indications.

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Evidence-based toolkit helps organizations reduce patient falls. Exploring strategies to operationalize cognitive reserve: A systematic review of reviews. Effects of PML and PML-RAR alpha antisense oligonucleotides on promyelocytic leukemia cell line NB4 Flow-mediated on-surface reconstitution of G-protein coupled receptors for applications in surface plasmon resonance biosensors. Such QR induction appears to protect breast cells against oestrogen-induced oxidative DNA damage, most likely by reducing reactive oestrogen metabolites termed catecholestrogen-quinones back to the hydroxy-catecholestrogens which may be conjugated.

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